Latest Posts by silver-griffincat - Page 10

constant drive to talk about my ocs but also the eternal issue of not being able to articulate my thoughts properly so just imagine that i am grabbing you like this. there are things in my mind but they are not escaping

？？？

get panini pressed, idiot

MY MOTHER USED MY PAYPAL TO MAKE A HUGE PURCHASE WITHOUT MY KNOWLEDGE OR PERMISSION; setting me back VERY BADLY in terms of money.

Donations & Awareness are appreciated NOW more than ever, please please PLEASE consider helping me, please.

PAY/PAL LINK

KO/FI LINK

I do not get fed at home, & on top of that my mother decided to actively give me a deadline to move out, please support me, any amount is so very highly appreciated.

I sat with a crying second grader today. (The age range is outside my wheelhouse but I was the most convenient adult.) He was crying, the other adults said, because his brother took a phone he was playing on. “Phone addicted,” everybody said. “If he would get up and play games with the other kids he wouldn’t be crying.”

He told me everyone lets his brother take things from him because his brother is younger, and doesn’t know better. He told me he doesn’t want to play because he’s tired, he has too many extracurriculars this summer and can’t get good sleep because “everyone in my camper is so loud when I’m trying to sleep.” He’s exhausted and only eight. His mom’s an acquaintance and told me she and the kid’s father are going through a separation — mom and four kids left the house to stay in a camper.

But people will seriously not listen to kids crying over seemingly minor things because on the surface it looks like a tantrum. If kids are given the space to articulate themselves they often will.

I’ve been seeing so many posts about burnout relating to working/studying hard......but burnout from fighting mental illnesses, disabilities, and chronic conditions needs to be talked about more. When you struggle with these things, you fight so many invisible battles a day that you don’t even realize you’re burnt out. You don’t have to be studying or working or “oN tHe gRiNd” to be burnt out. Being strong every day is the most grueling and exhausting kind of work. Please know that you are valid, even if you can’t get up and work/study. You are having to learn a curriculum that school doesn’t teach, and I’m so proud of you <33

Your moral ocd is lying to you and tumblr is lying to you. you do NOT have to reblog any post you dont want to and you dont need to justify it and youre not prejudiced against a certain marginalized group if you dont reblog an Upsetting Post about a Current Issue said group is facing

sunstone fluff for @lyss-butterscotch! pebbles gets shown the sky for the first time :)

In lineless even, since my line drawing skills have abandoned me for the time being

Cat colors genes masterpost

I copied all my descriptions from the cat color gene tournament here, with pictures and all. I often type out the genotypes, so this is a guide to what each letter means. Under the cut because it's very long.

Disclaimer: Sometimes I don't use the most common designations of a gene or an allele, and I'd like to apologize to everyone who's bothered by this. I have a very good reason for it: I like it better my way.

Genetics guide

Agouti (agouti signaling protein gene, ASIP): this gene determines whether the individual hairs will be banded or not.

dominant allele: A - banded hairs, tabby cat (wild type)

recessive allele: a - no bands, solid cat (variant)

A_ means if there's already an A allele, the other one can be either A or a.

A homozygous recessive cat with wild type alleles on every other gene will be solid black. Combined with other allele variants the a allele can produce other solid colors, different types of smokes and several more.

Actually there are more "secret" alleles for this gene: the agouti alleles of the asian leopard cat and other wild feline species. In certain hybrid breeds, most notably bengals, there's even a special coloration called charcoal born from the combination of the domestic cat's solid and the asian leopard cat's agouti allele.

Ticked (dickkopf wnt signaling pathway inhibitor 4, DKK4): this gene determines if there is any full-colored hairs, or only banded.

dominant allele(s - researchers found at least two): Ti - only banded hairs, ticked tabby cat (variant)

recessive allele: ti - some hairs don't have bands, "patterned" tabby cat (wild type)

If this is the only gene with variant allele, we'll have a black ticked tabby [black tabbies are also called brown, and other, mostly breed-specific names]. Ticked tabbies are possible in every color.

Nonagouti covers up the tickedness (this is called recessive epistasis): we won't see what a solid cat's genotype is on this gene. (Except when other genes make it possible. But that's biology for you.)

Spotted (?): this hypothetic gene can break up the tabby pattern's stripes into spots.

dominant allele: Sp - spotted tabby cat (variant)

recessive allele: sp - striped tabby cat (wild type)

Alone the Sp allele makes a black (brown ect) spotted tabby cat; of course, in combinations with other variant alleles, it can produce a wide variety of different colored spotted tabbies.

Both a and Ti covers up the spotted gene: its effect normally only visible on a cat with the A_ titi genotype.

Mackerel [i use the name mackerel since every other gene here is named after the dominant allele] (transmembrane aminopeptidase Q, Taqpep): this gene determines the type of the tabby pattern.

Blotched and classic are synonym terms.

dominant allele: TMc - narrow vertical lines, mackerel tabby cat (wild type)

recessive allele: tbl - wide, swirling lines, blotched or classic tabby cat (variant)

If every other gene is wild type except this, we'll have a black blotched tabby cat, but of course the tbl allele can produce lots of different colored classic tabbies.

All of the previously mentioned genes are able to nullify the effect of this one, so a mackerel or a blotched tabby must have A_ titi spsp genotype.

Additional annoyed remark: Despite the name, the so-called classic pattern is actually both the newer and the less common worldwide. My only guess for why it's named like that: it's the more common one in England. Well, thanks. (That's why I actually prefer the name blotched over classic.)

Brown (tyrosinase-related protein-1, TYRP1): this gene determines the quantity of the functional eumelanin.

dominant allele: B - full pigment production, black cat (wild type)

"middle" allele: b - less pigment, chocolate cat (variant)

recessive allele: bl- even less pigment, cinnamon cat (variant)

Order of dominance: B > b > bl

If every other allele is wild type except this, we'll have a chocolate or cinnamon mackerel tabby cat. (On the picture the cinnamon cat is spotted rather than striped, because i couldn't find a decent mackerel. So sad.) Chocolate and cinnamon cats are possible in every pattern.

Dilute (melanophilin, MLPH): this gene determines the distribution of the pigments.

dominant allele: D - even pigment distribution, dark cat (wild type)

recessive allele: d - clumped, uneven pigment distribution, diluted cat (variant)

black -> blue

chocolate -> lilac

cinnamon -> fawn

In these pictures the difference isn't that striking between the black and the blue mackerel tabby, but if you look up at the header, in solid cats it's much more pronounced.

For further comparison of undiluted and diluted color pairs on one picture (to eliminate differences in lightening):

black vs blue and red vs cream

Every possible color and pattern can be diluted (with the only exception of white).

Orange (?): this yet unidentified gene determines the type of the most prominent pigment: eumelanin on pheomelanin.

allele: O - mainly pheomelanin, red-based cat (variant)

allele: o - mainly eumelanin, black-based cat (wild type)

This gene is special in two related ways: first, it's located on the X chromosome, which means tomcats only have one allele; second, the alleles are codominant - if a cat carries both of them, it'll show both phenotypes: this is how we get tortoiseshell cats. This explains why almost all tortoiseshell cats are females - every tortie needs two different X chromosomes.

Combined with other variant alleles every possible color and pattern can occure as tortoiseshell, but the O allele is epistatic over a lot of genes: for example agouti (the phenotype of every orange cat is tabby, even the genetically solid ones) and brown (since eumelanin is mostly absent thus can't change - the genotypes OO B_, OO b_and OO blbl all mean red cat).

The dilute version of red is called cream.

The dilution level is always the same in the colors of a tortoiseshell: the undiluted black, chocolate and cinnamon is paired with red, the diluted blue, lilac and fawn are paired with cream.

White (receptor tyrosine kinase, KIT): this gene determines the size of the area the pigment producing cells (the melanocytes) reach.

dominant allele: W - basically no melanocytes, white cat (variant)

allele(s): ws - limited area is covered, white-spotted cat (variant)

allele: w - all of the body is covered by the melanocytes, full-colored cat (wild type)

recessive allele: wg - only the paws remain white, gloved cat (variant)

Order of dominance: W > ws = w > wg

Since the gloving allele is kind of specific to the breed, I used a birman cat to illustrate it.

Lots of alleles here! Actually I'm not sure what's the most accepted opinion about them, but since these variant are all mapped to KIT, I considered them alleles.

If every other gene shows the wild type except for this, we'll have a white, or a white-spotted black mackerel tabby cat, but thanks to the ws allele(s) every color and pattern can be combined with white patches. However, the W allele is epistatic over every other gene: if a cat has one or two copies of W, it will be white regardless everything else.

ws is interesting: it has an additive effect, a cat with the wsws genotype will have more white than a cat with only one copy of it.

wg is fully recessive: the gloved phenotype only present if the cat's genotype is wgwg.

Color restriction (tyrosinase, TYR): mutations on this gene will result in temperature-sensitivity in the pigment production, the cats will be lighter on the warm and darker on the cooler areas of their bodies.

dominant allele: C - regular pigment production, full colored cat (wild type)

allele: cb - moderately reduced pigment production: burmese color restriction, sepia cat (variant)

allele: cm - reduced pigment production, bangkok color restriction, mocha cat (variant)

allele: cs - highly reduced pigment production: siamese color restriction, pointed cat (variant)

recessive allele: c - no pigment production, albino cat (variant)

Dominance order: C > cb = cm = cs > c

Now this group is a lot. Not only five different alleles (mocha was found relatively recently in Thailand), but the middle three are all intermediate with each other meaning that actually we have eight different phenotypes (illustration from messybeast; full color and albino are absent):

I used solid cats for illustration, because in the thai breed (the cats i used belong to this) they are often preferred over tabbies, so it's easier to find pictures; also, it's much more simple to compare them.

(Photos from The Thai Cat Center and Bangkok Mocha Cat, and Pangur from @pangur-and-grim as an albino cat)

Please note that all of these varieties are very changeable; the pictures (especially those of the heterozygotes) are far from representing all cats carrying the respective genotypes.

Alone these variants makes some type of a black (seal) mackerel tabby point cat, but every type of color restriction can occure together with all possible colors and patterns.

***The color restriction gene won the Cat Color Gene Tournament!***

Inhibitor (?): this unidentified gene reduces the pheomelanin production, thus removes the warm tones of the fur (the hairs have white-black banding instead of yellow-black).

dominant allele: I - reduced pheomelanin, cooler toned cat (variant)

recessive allele: i - normal pheomelanin, warmer toned cat (wild type)

If every other allele is wild type except for this, we'll have a black silver mackerel tabby cat. Combined with other alleles it can produce lots of different silver (tabby) and smoke (solid) varieties.

Wide band (?): This hypothetic gene makes the yellow bands on the agouti hairs wider, resulting in a lighter, yellowish pelt. Based on the width of the pale bands we can differentiate between golden (middle band width) and shaded (maximal band width, color is pushed up into the tip).

dominant allele: Wb - reduced area of eumelanin, warmer toned cat (variant)

recessive allele: wb - normal area of eumelanin, cooler toned cat (wild type)

Golden is quite a mess; right now there is one identified gene (found first in siberians), but persians and many more breeds must have different gene(s), based on the interaction with the inhibitor gene (siberian golden + silver = bimetallic, persian golden + silver = silver shaded or chinchilla), and the inheritance patterns (the siberian alleles are recessive, while persian golden appears to be dominant). Since lots of breeds allow golden, and sometimes it can be found even in stray cats, I say who knows what genes and alleles are out there! This is all a hardly understood, very exciting and currently researched area.

If every other gene stays wild type except for this, we'll have a black golden mackerel tabby or a black golden shaded cat. Combined with other alleles it can produce lots of different golden and silver varieties.

Low-grade white (?): Again, hypothetic gene(s). Even with the extreme variability of the white spotting allele(s), the existence of some independently inherited genes is strongly suspected. Their effects most commonly manifest as a white locket: a small white patch on the chest or the belly, and/or a white tail tip. I'm not sure if there is any consensus whether these are more likely to be recessive or dominant alleles.

Dilute modifier (?): This unidentified gene changes the color of a diluted cat, the coloration becomes more brownish.

dominant allele: Dm - (variant)

recessive allele: dm - (wild type)

blue, lilac, fawn -> caramel

cream -> apricot

I put here a cat in all three diluted colors to compare them with the caramel tabby. It's hard to spot the differences, isn't it?

Since this is a dilute modifier, the D allele covers it, and we can only see its effect on cats with dd genotype.

It can be found only in a few breeds: orientals (including related breeds), burmese, different rexes. To our current knowledge, of course.

Extension (melanocortin 1 receptor, MC1R): This gene replaces eumelanin with pheomelanin resulting in a yellowish or reddish furred cat. The change often happens gradually during the first years of the cat's life.

dominant allele: E - eumelanin remains, black adjacent cat (wild type)

recessive alleles: e, er, ec - pheomelanin takes over, yellow/red adjacent cat: amber, russet or serdolik (variant)

All three recessive variants are new mutations found recently in different breeds: the color amber in the 1990s in norwegian forest cats, the color russet in 2007 in burmese, and the color carnelian or serdolik in 2018 in kurilian bobtails (at least that's the first mention). We don't know anything about their interactions, or their effects on cats outside of their respective breeds.

The gene only effects eumelanin, so the O allele is epistatic over the it. However, because of the properties of the overpowering pheomelanin, every e allele is epistatic over agouti, so the tabby patterns will show up on aa cats as well.

Wide band (serine peptidase, CORIN): This hypothetic gene makes the yellow bands on the agouti hairs wider, resulting in a lighter, yellowish pelt.

dominant allele: Wb - eumelanin on normal sized area, darker cat (wild type)

recessive alleles: wbSIB, wbeSIB, wbBRI - eumelanin on reduced area, lighter cat (variant)

Ohhh, citizens of tumblr, we're really in it now. So. In the moment, we have, I believe, three mutations found on this gene: the sunshine (wbSIB) and extreme sunshine (wbeSIB) in the siberian breed, and the copper (wbBRI) in british cats. (I only show the sunshine and the copper here.) The novelty of these mutations means that the breeders still often call them simply golden instead of the new names, so it's difficult to find reliable data. Further complicating the situation, most likely both breeds have more wide band gene(s) beyond CORIN, and especially the copper cat above is the result of the combination of several wb genes.

Karpati (?): This unidentified gene makes the extremeties (face, ears, legs, tail) white kinda like a reverse colorpoint cat, and causes a roaning effect: scatters white hairs everywhere on the body.

dominant allele: K - whited extremities, karpati cat (variant)

recessive alleles: k - normal pigmant production, full colored cat (wild type)

Karpati seems to show intermediate inheritance with significantly more white on a homozygous then a heterozygote cat. This gene is studied for a very short time, and mostly on heterozygotes since they are much more common. The cats appearence changes during their life and also with the seasons: they born very similar to a fever coated kitten but with white ears, then to the end of their first year they almost completely lose the white (at least the heterozygous cats - the homozygotes become darker but still keep strange white patterns), then slowly gain it back as they age.

The karpati mutation is present in the stray cat population in middle-east Europe (including Hungary where I live, wahoo! and indeed, I can regularly see one or two karpaties in facebook adoptions groups and such). It's also introduced to some established breeds (LaPerm, Sphynx ect) and the creation of its own breed also began under the Transylvanian name.

splatoon ring pops

splatoon ring pops

either seven red suns or dozen of trees (iterator oc of mine) in splatoon 3.

definitely weird (serious game + none-serious game), but a manageable situation either way.

The last character you drew/wrote about is now stuck in the last game you played. How screwed are they?

pebbles embodied this meme for a minute there

could you draw fp getting a group hug from suns and moon and nsh please? if you're ok with that! i think he deserves lots of hugs and it would be rather heartwarming ^^

Double ask @tanyabadtime159

Anyways hi hello im back sorry i think something possessed me to go insane for abit there. Back to your regularly scheduled doodle asks ^-^

And yeah i agree Pebbles deserves a lil bit more love after what he's been through

new favorite image: all 2,700 possible tropical fish variants in minecraft

I turned it into a man

pebble

attack kill kill kill

looking at moon's situation, I think it's possible. at least to an extent.

with her umbilical cable being completely severed, her having been mostly reduced to her puppet, etc.

the issue with the whole freedom thing is that the puppet needs at least some connection to the can to survive. if moon's can were dead, or her puppet entirely disconnected, she wouldn't be there.

but she is alive, despite the severed cable.

so I think the explanation and solution here is the weak wireless connection she seems to have with, yk, the rest of herself. so, hypothetically, a freed puppet would be able to roam however far that connection reaches (as long as power isn't an issue, and they're taking a few neurons along with them).

well, that's if they're happy with either crawling around like some sort of feral creature, or tediously learning to walk on feet that weren't made for it, and can accept that they may be unable to directly communicate with their can ever again unless they're able to reattach themselves.

I don't think the arm would be much of an issue. with the functions it seems to have, they could just as well be performed by something else.

and that's that. but there might be a completely different way, that technically doesn't even involve getting out. some advanced drone shenanigans, perhaps.

hey

hypothetically

for no particular reason at all

how do you think an iterator would Actually be able to Go Off The String. because as fun as it is to imagine them just prying off the umbilical learning to walk and wandering away, i don't think the puppet would be able to survive without the umbilical (power source and whatnot)

so How Would It Be Achieved

go wild as long as it's like. even slightly plausible. i'm lookin for raw ideas

my life changed on the day I discovered low effort cooking for myself.

instead of spending hours on an elaborate dish, I can just throw a bunch of stuff in salty water or between two slices of bread, cook/fry that bad boy, and boom. I got myself some food. I'm cooking way more often than I used to.

Second half of the text underneath

I am a huge fan of biology (even if still new to it), and despite all the jokes people make, nobody's very far off on the Rot- and I'm amazed that even in-game evidence points to it too.

Though I think other interpretations are real neat as well! Think o this more of an analysis for fun than concrete lore stuff whatever.

this damned glorious concept has been bouncing in my head like a screensaver for a little while now.

so here's suns being at least mildly disturbed at the sight of their own limp lifeless body.

the idea is just too good to not be expanded on, ain't it.

disease acquired

I cannot count how many times I've watched this over the last few days.

aint no rain's gonna break my stride

for anyone who has splatoon 3, here's a replay code of the least serious turf war match I had in a long time. it's not really a squid party, just everyone collectively deciding to not really try. posting this because I thought it was funny.

here, have a sliver of straw. as a treat.